Li Z B, Chen H Y, Li J Y, et al. Clinical, laboratory and imaging features of high altitude pulmonary edema in Tibetan Plateau[J]. Chin Med Sci J, 2018, 33(3):160-173.
[3]
Swenson E R. Early hours in the development of high-altitude pulmonary edema: time course and mechanisms[J]. J Appl Physiol (1985), 2020, 128(6):1539-1546.
Luks A M, Auerbach P S, Freer L, et al. Wilderness medical society clinical practice guidelines for the prevention and treatment of acute altitude illness: 2019 update [J]. Wilderness Environ Med, 2019, 30(4S):S3-S18.
[6]
Zhang X, Xie L, Long J, et al. Salidroside: A review of its recent advances in synthetic pathways and pharmacological properties[J]. Chem Biol Interact, 2021, 339:109268.
[7]
Wang N, Song J, Zhou G, et al. Mechanism of salidroside relieving the acute hypoxia-induced myocardial injury through the PI3K/Akt pathway[J]. Saudi J Biol Sci, 2020, 27(6):1533-1537.
[8]
Nan X, Su S, Ma K, et al. Bioactive fraction of rhodiola algida against chronic hypoxia-induced pulmonary arterial hypertension and its anti-proliferation mechanism in rats[J]. J Ethnopharmacol, 2018, 216:175-183.
[9]
Li Y, Zhang Y, Zhang Y. Research advances in pathogenesis and prophylactic measures of acute high altitude illness[J]. Respir Med, 2018, 145:145-152.
[10]
Young J M, Williams D R, Thompson A. Thin air, thick vessels: historical and current perspectives on hypoxic pulmonary hypertension[J]. Front Med (Lausanne), 2019, 6:93.
[11]
Smadja D M, Mauge L, Sanchez O, et al. Distinct patterns of circulating endothelial cells in pulmonary hypertension[J]. Eur Respir J, 2010, 36(6):1284-1293.
[12]
Wang T, Hou J, Xiao W, et al. Chinese medicinal plants for the potential management of high-altitude pulmonary oedema and pulmonary hypertension[J]. Pharm Biol, 2020, 58(1):815-827.
[13]
Chang P K, Yen I C, Tsai W C, et al. Protective effects of rhodiola crenulata extract on hypoxia-induced endothelial damage via regulation of AMPK and ERK pathways[J]. Int J Mol Sci, 2018, 19(8):1435-1442.
[14]
Soree P, Gupta R K, Singh K, et al. Raised HIF1α during normoxia in high altitude pulmonary edema susceptible non-mountaineers[J]. Sci Rep, 2016, 6: 26468.
Sharma K R, Mishra R, Gautam J, et al. Patchy vasoconstriction versus inflammation: a debate in the pathogenesis of high altitude pulmonary edema [J]. Cureus, 2020, 12(9):e10371.
[21]
Gojkovic M, Darmasaputra G S, Velişa P, et al. Deregulated hypoxic response in myeloid cells: A model for high-altitude pulmonary oedema (HAPE)[J]. Acta Physiol (Oxf), 2020, 229(2): e13461.
[22]
关 爽.红景天苷的抗炎作用及其对炎症信号转导通路的调控[D].长春:吉林大学,2011.
[23]
Gambhir S, Vyas D, Hollis M, et al. Nuclear factor kappa B role in inflammation associated gastrointestinal malignancies [J]. World J Gastroenterol, 2015, 21(11):3174-3183.
[24]
You L, Zhang D, Geng H, et al. Salidroside protects endothelial cells against LPS-induced inflammatory injury by inhibiting NLRP3 and enhancing autophagy[J]. BMC Complement Med Ther, 2021, 21(1):146.
[25]
Hu R, Wang M Q, Ni S H, et al. Salidroside ameliorates endothelial inflammation and oxidative stress by regulating the AMPK/NF-κB/NLRP3 signaling pathway in AGEs-induced HUVECs[J]. Eur J Pharmacol, 2020, 867:172797.
[26]
Cai W, Liu Z, Li G, et al. The effects of a graded increase in chronic hypoxia exposure duration on healthy rats at high-altitude [J]. Int J Clin Exp Pathol, 2019, 12(6):1975-1991.
[27]
Cai W, Liu S, Liu Z, et al. Downregulation of lung miR-203a-3p expression by high-altitude hypoxia enhances VEGF/Notch signaling[J]. Aging (Albany NY), 2020, 12(5): 4247-4267.
[28]
Lee S Y, Li M H, Shi L S, et al. Rhodiola crenulata extract alleviates hypoxic pulmonary edema in rats [J]. Evid Based Complement Alternat Med, 2013, 2013:718739.
Magnani N D, Dada L A, Queisser M A, et al. HIF and HOIL-1L-mediated PKCζ degradation stabilizes plasma membrane Na, K-ATPase to protect against hypoxia-induced lung injury[J]. Proc Natl Acad Sci U S A, 2017, 114(47):E10178-E10186.
[31]
谭春燕.红景天苷抗高原缺氧作用研究[D]. 拉萨:西藏大学, 2015.
[32]
Zhu Z, Li J, Zhang X. Salidroside protects against ox-LDL-induced endothelial injury by enhancing autophagy mediated by SIRT1-FoxO1 pathway [J]. BMC Complement Altern Med, 2019, 19(1):111.