脾功能亢进对肝纤维化影响的研究进展

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[1]	Takaaki H,Scott L. Friedman, Y H, et al. Hepatic stellate cells as key target in liver fibrosis [J]. Adv Drug Deliv Rev, 2017, 121(1): 27-42.
[2]	Friedman S L, Roll F J, Boyles J, et al. Hepatic lipocytes: The principal collagen-producing cells of normal rat liver[J]. Proc Natl Acad Sci, 1985, 82(24):8681-8685.
[3]	Pellicoro A. Liver fibrosis and repair: immune regulation of wound healing in a solid organ[J]. Nat Rev Immunol, 2014, 14(3): 181-194.
[4]	Barnes M A. Macrophage migration inhibitory factor contributes to ethanol-induced liver injury by mediating cell injury, steatohepatitis, and steatosis[J]. Hepatology, 2013,57(5): 1980-1991.
[5]	Ide M, Kuwamura M,Kotani T, et al. Effects of aadolinium chloride (GdCl3) on the appearance of macrophage populations and fibrogenesis in thioacetamide-induced rat hepatic lesions[J]. J Comp Pathol, 2005, 133(2-3): 92-102.
[6]	Duffield J S, Forbes S J, Constandinoa C M, et al. Selective depletion of macrophages reveals distinct, opposing roles during liver injury and repair[J]. J Clin Invest, 2005,115(1): 56-65.
[7]	Ramachandran P, Pellicoro A, Madeleine A, et al. Differential Ly-6C expression identifies the recruited macrophage phenotype, which orchestrates the regression of murine liver fibrosis[J]. Proc Natl Acad Sci, 2012, 109(46): E3186-E3195.
[8]	Holt M P, Cheng L L, Ju C, et al. Identification and characterization of infiltrating macrophages in acetaminophen‐induced liver injury[J]. J Leukoc Biol, 2008, 84(6): 1410-1421.
[9]	Michael A G, Alison C M, Prakash R, et al. Ly6C~(hi) monocytes direct alternatively activated profibrotic macrophage regulation of lung fibrosis[J]. Am J Respir Crit Care Med, 2011, 184(5): 569-581.
[10]	Shahrouz G, Rohan V, Tyler R,et al. Platelet TGF-b1deficiency decreases liver fibrosis in a mouse model of liver injury[J]. Blood Adv, 2018 ,2(5): 470-480.
[11]	Kempiński R, Neubauer K, Poniewierka E, et al. The immunoreactivity of TGF-β1 in non-alcoholic fatty liver disease[J]. Folia Histochem Cytobiol, 2019, 57(2): 74-83.
[12]	Tomonori A, Kyoko K A, Akira U, et al. Spleen-derived lipocalin-2 in the portal vein regulates Kupffer cells activation and attenuates the development of liver fibrosis in mice[J]. Lab Invest, 2017, 97(8): 890-902.
[13]	Yada A, Limuro Y, Uyama N, et al. Splenectomy attenuates murine liver fibrosis with hypersplenism stimulating hepatic accumulation of Ly-6Clo macrophages[J]. J Hepatol, 2015, 63(4): 905-916.
[14]	Nomura Y, Kage M, Ogta T,et al. Influence of splenectomy in patients with liver cirrhosis and hypersplenism[J]. Hepatol Res 2014, 44(10): E100-E109.
[15]	Herro R,Croft M. The control of tissue fibrosis by the inflammatory molecule LIGHT (TNF Superfamily member 14)[J]. Pharmacol Res, 2016, 2(104): 151-155.
[16]	Herro R, Tamada K, Croft M, et al. The tumor necrosis factor superfamily molecule LIGHT promotes keratinocyte activity and skin fibrosis[J]. J Invest Dermatol, 2015, 135(8): 2109-2118.
[17]	DohertyT A, Soroosh P, Khorram N,et al. The tumor necrosis factor family member LIGHT is a target for asthmatic airway remodeling[J]. Nat Med, 2011, 17(5): 596-603.
[18]	Herro R, Tamada K, Croft M, et al. Tumor necrosis factor superfamily 14 (LIGHT) controls thymic stromal lymphopoietin to drive pulmonary fibrosis[J]. J Allergy Clin Immunol, 2015, 136(3): 757-768.
[19]	BashourF N, Teran J C,Mullen KD. Prevalence of peripheral blood cytopenias (hypersplenism) in patients with nonalcoholic chronic liver disease[J]. Am J Gastroenterol, 2000,95(10): 2936-2939.
[20]	NomuraY, Kage M, Ogata T, et al. Influence of splenectomy in patients with liver cirrhosis and hypersplenism[J]. Hepatol Res, 2014. 44(10): E100-E109.
[21]	Jiang A, Shu Z, Zongfang L, et al. miR-615-3p promotes the phagocytic capacity of splenic macrophages by targeting ligand-dependent nuclear receptor corepressor in cirrhosis-related portal hypertension[J]. Exp Biol Med, 2011, 236(6): 672-680.
[22]	Song R, Shu Z, Manxiang L , et al. NF-kappa B p65 and c-Rel subunits promote phagocytosis and cytokine secretion by splenic macrophages in cirrhotic patients with hypersplenism[J]. Int J Biochem Cell Biol, 2013, 45(2): 335-343.
[23]	Knutson M,Wessling R M. Iron metabolism in the reticuloendothelial system[J]. Crit Rev Biochem Mol Biol, 2003, 38(1): 61-88.
[24]	Tarantino G.Liver-spleen axis: Intersection between immunity, infections and metabolism[J]. World J Gastroenterol, 2013, 19(23): 3534-3542.
[25]	Lee Y A, Wallace MC, Friedman S L. Pathobiology of liver fibrosis: a translational success story[J]. Gut, 2015, 64(5): 830-841.
[26]	贾冬威. RACK1在肝纤维化调控中的功能和机制以及PQQ干预肝纤维化发生发展的研究[D].上海:复旦大学,2013.
[27]	Akahoshi T, Hashizume T, Tanoue K, et al. Role of the spleen in liver fibrosis in rats may be mediated by transforming growth factor β-1[J]. J Gastroenterol Hepatol, 2002, 17(1): 59-65.
[28]	Asanoma M, Ikemoto T, Mori H, et al. Cytokine expression in spleen affects progression of liver cirrhosis through liver-spleen cross-talk[J]. Hepatol Res, 2014, 44(12): 1217-1223.
[29]	汪家富,刘绪舜. 脾脏及脾切除对肝硬化影响的研究进展[J]. 肝胆胰外科杂志,2012,24(5): 438-441.
[30]	Mauri D N, Ebner R, Montgomery R I, et al. LIGHT, a new member of the TNF superfamily, and lymphotoxin alpha are ligands for herpesvirus entry mediator[J]. Immunity, 1998, 8(1): 21-30.
[31]	Herro R, Antunes R D, Aguilera A R, et al. Tumor necrosis factor superfamily 14 (LIGHT) controls thymic stromal lymphopoietin to drive pulmonary fibrosis[J]. J Allergy Clin Immunol, 2015, 136(3): 757-768.
[32]	Antunes D S, Amit K M, Lisa M, et al. TNFSF14 (LIGHT) exhibits inflammatory activities in lung fibroblasts complementary to IL-13 and TGF-β[J]. Front Immunol, 2018, 9(19): 576-587.