病毒性心肌炎自身免疫反应期发病机制的研究进展

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[1]	江国强, 刘政. 急性重症病毒性心肌炎22 例[J]. 武警医学, 2012,23(3): 240-241.
[6]	Kallwellis-Opara A, Drner A, Poller W C, et al. Autoimmunological features in inflammatory cardiomyopathy[J]. Clin Res Cardiol, 2007,96(7):469-480.
[7]	Fairweather D, Petri M A, Coronado M J, et al. Autoimmune heart disease: role of sex hormones and autoantibodies in disease pathogenesis[J]. Expert Rev Clin Immunol, 2012,8(3):269-284.
[2]	Reddy J, Massilamany C, Buskiewicz I, et al. Autoimmunity in viral myocarditis[ J]. Curr Opin Rheumatol, 2013,25(4):502-508.
[8]	Rose N R. Critical cytokine pathways to cardiac inflammation[J]. J Interferon Cytokine Res, 2011,31(10):705-710.
[9]	Shijie Z, Moriya J, Yamakawa J, et al. Mao-to Prolongs the survival of and reduces TNF-alpha expression in mice with viral myocarditis[J]. Evid Based Complement Alternat Med, 2010,7(3):341-349.
[3]	龚非力. 医学免疫学[M]. 2版. 北京:科学出版社, 2007:141-151.
[10]	Kara E E, Comerford I, Bastow C R, et al. Distinct chemokine receptor axes regulate Th 9 cell trafficking to allergic and autoimmune inflammatory sites[J]. J Immunol, 2013,191(3):1110-1117.
[4]	Fairweather D, Stafford K A, Sung Y K. Update on coxsackievirus B3 myocarditis[J]. Curr Opin Rheumatol, 2012,24(4):401-407.
[5]	Noutsias M, Patil V J, Maisch B. Cellular immune mechanisms in myocarditis[J]. Herz, 2012,37(8):830-835.
[11]	Veldhoen M, Uyttenhove C, van Snick J, et al. Transforming growth factor-beta‘reprograms’ the differentiation of T helper 2 cells and promotes an interleukin 9-producing subset[J].Nat Immunol, 2008,9(12):1341-1346.
[6]	Kallwellis-Opara A, Drner A, Poller W C, et al. Autoimmunological features in inflammatory cardiomyopathy[J]. Clin Res Cardiol, 2007,96(7):469-480.
[12]	Singh T P, Schn M P, Wallbrecht K, et al. Involvement of IL-9 in Th17-associated inflammation and angiogenesis of psoriasis[J]. Plos One, 2013,8(1):51752.
[7]	Fairweather D, Petri M A, Coronado M J, et al. Autoimmune heart disease: role of sex hormones and autoantibodies in disease pathogenesis[J]. Expert Rev Clin Immunol, 2012,8(3):269-284.
[13]	Qing K, Weifeng W, Fan Y, et al. Distinct different expression of Th17 and Th9 cells in coxsackie virus B3-induced mice viral myocarditis[J]. Virol J, 2011,8(1):267-269.
[8]	Rose N R. Critical cytokine pathways to cardiac inflammation[J]. J Interferon Cytokine Res, 2011,31(10):705-710.
[9]	Shijie Z, Moriya J, Yamakawa J, et al. Mao-to Prolongs the survival of and reduces TNF-alpha expression in mice with viral myocarditis[J]. Evid Based Complement Alternat Med, 2010,7(3):341-349.
[10]	Kara E E, Comerford I, Bastow C R, et al. Distinct chemokine receptor axes regulate Th 9 cell trafficking to allergic and autoimmune inflammatory sites[J]. J Immunol, 2013,191(3):1110-1117.
[11]	Veldhoen M, Uyttenhove C, van Snick J, et al. Transforming growth factor-beta‘reprograms’ the differentiation of T helper 2 cells and promotes an interleukin 9-producing subset[J].Nat Immunol, 2008,9(12):1341-1346.
[14]	杨帆, 伍伟锋, 颜玉鸾, 等. 病毒性心肌炎小鼠IL-9＋/Th17细胞的变化[J]. 临床心血管病杂志, 2013,29(8):624-627.
[12]	Singh T P, Schn M P, Wallbrecht K, et al. Involvement of IL-9 in Th17-associated inflammation and angiogenesis of psoriasis[J]. Plos One, 2013,8(1):51752.
[15]	Baldeviano G C, Barin J G, Talor M V, et al. Interleukin-17A is dispensable for myocarditis but essential for the progression to dilated cardiomyopathy[J]. Circ Res, 2010,106(10):1646-1655.
[13]	Qing K, Weifeng W, Fan Y, et al. Distinct different expression of Th17 and Th9 cells in coxsackie virus B3-induced mice viral myocarditis[J]. Virol J, 2011,8(1):267-269.
[16]	Yuan J, Yu M, Lin Q W, et al. Th17 cells contribute to viral replication in coxsackievirus B3-induced acute viral myocarditis [J].J Immunol, 2010,185(7):4004-4010.
[14]	杨帆, 伍伟锋, 颜玉鸾, 等. 病毒性心肌炎小鼠IL-9＋/Th17细胞的变化[J]. 临床心血管病杂志, 2013,29(8):624-627.
[17]	Bauquet A T, Jin H, Paterson A M, et al. The costimulatory molecule ICOS regulates the expression of c-Maf and IL-21 in the development of follicular T helper cells and TH-17 cells[J]. Nat Immunol, 2009,10(2):167-175.
[18]	Liu Y, Pan W, Yang S, et al. Interleukin-22 protects rat PC12 pheochromocytoma cells from serum deprivation-induced cell death[J]. Mol Cell Biochem, 2012,371(1-2):137-146.
[15]	Baldeviano G C, Barin J G, Talor M V, et al. Interleukin-17A is dispensable for myocarditis but essential for the progression to dilated cardiomyopathy[J]. Circ Res, 2010,106(10):1646-1655.
[19]	Chang H, Hanawa H, Liu H, et al. Hydrodynamic-based delivery of an interleukin-22-Ig fusion gene ameliorates experimental autoimmune myocarditis in rats[J]. J Immunol, 2006,177(6):3635-3643.
[16]	Yuan J, Yu M, Lin Q W, et al. Th17 cells contribute to viral replication in coxsackievirus B3-induced acute viral myocarditis [J].J Immunol, 2010,185(7):4004-4010.
[20]	Kong Q, Wu W, Yang F, et al. Increased expressions of IL-22 and Th22 cells in the coxsackievirus B3-Induced mice acute viral myocarditis[J]. Virol J, 2012,9:232-234.
[17]	Bauquet A T, Jin H, Paterson A M, et al. The costimulatory molecule ICOS regulates the expression of c-Maf and IL-21 in the development of follicular T helper cells and TH-17 cells[J]. Nat Immunol, 2009,10(2):167-175.
[18]	Liu Y, Pan W, Yang S, et al. Interleukin-22 protects rat PC12 pheochromocytoma cells from serum deprivation-induced cell death[J]. Mol Cell Biochem, 2012,371(1-2):137-146.
[19]	Chang H, Hanawa H, Liu H, et al. Hydrodynamic-based delivery of an interleukin-22-Ig fusion gene ameliorates experimental autoimmune myocarditis in rats[J]. J Immunol, 2006,177(6):3635-3643.
[20]	Kong Q, Wu W, Yang F, et al. Increased expressions of IL-22 and Th22 cells in the coxsackievirus B3-Induced mice acute viral myocarditis[J]. Virol J, 2012,9:232-234.