[1] |
Harrison S, Geppetti P. Substance p [J]. Int J Biochem Cell Biol, 2001, 33(6):555-576.
|
[2] |
Sarathi V, Lila A R, Bandgar T R, et al. Pheochromocytoma and pregnancy: a rare but dangerous combination[J]. Endocr Pract, 2010, 16(2):300-309.
|
[3] |
Lénárd L, László K, Kertes E, et al. Substance P and neurotensin in the limbic system: Their roles in reinforcement and memory consolidation.[J]. Neurosci Biobehav Rev, 2018,85:1-20.
|
[4] |
Sun J, Bhatia M. Substance P at the neuro-immune crosstalk in the modulation of inflammation, asthma and antimicrobial host defense.[J].Inflamm Allergy Drug Targets, 2014, 13(2):112-120.
|
[5] |
Aicher S A, Punnoose A, Goldberg A. Muopioid receptors often colocalize with the substance P receptor (NK1) in the trigeminal dorsal horn [J]. J Neurosci, 2000, 20(11):4345-4354.
|
[6] |
Parenti C, Aricò G, Ronsisvalle G, et al. Supraspinal injection of substance P attenuates allodynia and hyperalgesia in a rat model of inflammatory pain [J]. Peptides, 2012, 34(2):412-418.
|
[7] |
Li W W, Guo T Z, Shi X, et al. Substance P spinal signaling induces glial activation and nociceptive sensitization after fracture [J]. Neuroscience, 2015, 310:73-90.
|
[8] |
Teodoro F C, Tronco Júnior M F, Zampronio A R, et al. Peripheral substance P and neurokinin-1 receptors have a role in inflammatory and neuropathic orofacial pain models.[J]. Neuropeptides, 2013, 47(3):199-206.
|
[9] |
Woolf C J, Max M B. Mechanism-based pain diagnosis: issues for analgesic drug development [J]. Anesthesiology, 2001, 95(1):241-249.
|
[10] |
Chen K, Zhang Z F, Liao M F, et al. Blocking PAR2 attenuates oxaliplatin-induced neuropathic pain via TRPV1 and releases of substance P and CGRP in superficial dorsal horn of spinal cord[J]. J Neurol Sci, 2015, 352(1-2):62-67.
|
[11] |
Chiba T, Oka Y, Kambe T, et al. Paclitaxel-induced peripheral neuropathy increases substance P release in rat spinal cord[J]. Eur J Pharmacol, 2016, 770:46-51.
|
[12] |
Nakayama T, Naono R, Ikeda T, et al. NMDA and AMPA receptors contribute to the maintenance of substance P-induced thermal hyperalgesia [J]. Neurosci Res, 2010, 67(1):18-24.
|
[13] |
Khasabov S G, Malecha P, Noack J, et al. Hyperalgesia and sensitization of dorsal horn neurons following activation of NK-1 receptors in the rostral ventromedial medulla[J]. J Neurophysiol, 2017, 118(5):jn.00478.2017.
|
[14] |
Uematsu T, Sakai A, Ito H, et al. Intra-articular administration of tachykinin NK1receptor antagonists reduces hyperalgesia and cartilage destruction in the inflammatory joint in rats with adjuvant-induced arthritis.[J]. Eur J Pharmacol, 2011, 668(1-2):163.
|
[15] |
Borbély É, Sándor K, Markovics A, et al. Role of capsaicin-sensitive nerves and tachykinins in mast cell tryptase-induced inflammation of murine knees [J]. Inflamm Res, 2016, 65(9):725-736.
|
[16] |
赵国敏, 尹金淑. P物质及其受体神经激肽1受体与疼痛的相关性研究[J]. 医学综述, 2015, 21(16): 2890-2893.
|
[17] |
Li Z Y, Yang Y T, Hong J, et al. Extracellular signal-regulated kinase, substance P and neurokinin-1 are involved in the analgesic mechanism of herb-partitioned moxibustion[J]. Neural Regen Res, 2017, 12(9):1472-1478.
|
[18] |
项红兵,田玉科. 星形胶质细胞与疼痛敏化调控[J]. 临床麻醉学杂志,2003, 9:576-577.
|
[19] |
Jinbo L, Lijian D. PERK pathway is involved in oxygen-glucose-serum deprivation-induced NF-kB activation via ROS generation in spinal cord astrocytes[J]. Biochem Biophys Res Commun, 2015, 467(2):197-203.
|
[20] |
Lagerstrã M M C, Rogoz K, Abrahamsen B, et al. A sensory subpopulation depends on vesicular glutamate transporter 2 for mechanical pain, and together with substance P, inflammatory pain [J]. Proc Natl Acad Sci U S A, 2011, 108(14):5789-5794.
|
[21] |
Woolf C J; American College of Physicians; American Physiological Society. Pain: moving from symptom control toward mechanism-specific pharmacologic management.[J]. Ann Intern Med, 2004, 140(6): 441-451.
|
[22] |
徐 良, 裘 涛, 张丽娟,等. 慢性压迫损伤性神经病理性疼痛大鼠模型中脊髓背角ERK、CREB、BDNF表达的变化[J]. 浙江医学, 2016, 38(16):1341-1344.
|
[23] |
Nakamura Y, Izumi H, Fukushige R, et al. Continuous infusion of substance P into rat striatum alleviates nociceptive behavior via phosphorylation of extracellular signal-regulated kinase 1/2[J]. J Neurochem, 2014, 131(6):755-766.
|
[24] |
Kawasaki Y, Kohno T, Zhuang Z Y, et al. Ionotropic and metabotropic receptors, protein kinase A, protein kinase C, and Src contribute to C-fiber-induced ERK activation and cAMP response element-binding protein phosphorylation in dorsal horn neurons, leading to central sensitization[J]. J Neurosci, 2004, 24(38):8310.
|
[25] |
Wei F, Vadakkan K I, Toyoda H, et al. Calcium calmodulin-stimulated adenylyl cyclases contribute to activation of extracellular signal-regulated kinase in spinal dorsal horn neurons in adult rats and mice[J]. J Neurosci, 2006, 26(3):851-861.
|
[26] |
刘春芳,姜 虹.右旋美托咪啶对切口痛大鼠脊髓背角神经元型一氧化氮合酶和P物质mRNA表达的影响[J].上海医学, 2009, 32(6): 483-486.
|
[27] |
Komatsu T. Study of supplementary analgesics capable of reducing the dosage of morphine[J].Yakugaku Zasshi, 2016, 136(2):329.
|
[28] |
习 亮,梁 伟,张永峰.NF-κB信号通路抑制剂对P物质作用后脊髓星形胶质细胞GFAP、炎性因子表达的影响[J].山东医药, 2017, 57(44): 45-47.
|
[29] |
Etemadi L, Pettersson L M E, Danielsen N. UVB irradiation induces rapid changes in galanin, substance P and c-fos immunoreactivity in rat dorsal root ganglia and spinal cord[J]. Peptides, 2016, 87: 71-83.
|
[30] |
Yang X, Liu J, Liu Z J, et al. Reversal of bone cancer pain by HSV-1-mediated silencing of CNTF in an afferent area of the spinal cord associated with AKT-ERK signal inhibition [J]. Curr Gene Ther, 2014, 14(5):377.
|
|
|