单核巨噬细胞特点及其盐敏感高血压靶器官损伤机制

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[1]	Benjamin E J, Blaha M J,Chiuve S E, et al. Heart disease and stroke statistics-2017 update: a report from the American Heart Association[J]. Circulation,2017,135(10):e146-e603.
[2]	杨国红,周欣,姜铁民,等. 高血压发病机制的基因学研究进展[J]. 武警医学,2013,24(11):685-688.
[3]	Mimran A, du Cailar G. Dietary sodium: the dark horse amongst cardiovascular and renal risk factors[J]. Nephrol Dial Transplant,2008,23(7):2138-2141.
[4]	Varagic J,Frohlich E D,Susic D, et al. AT1 receptor antagonism attenuates target organ effects of salt excess in SHRs without affecting pressure[J]. Am J Physiol Heart Circ Physiol,2008,294(2):H853-858.
[5]	Zhang X,Li H,Li Y, et al. Increased circulating Ly6Chigh monocyte subsets are correlated with cerebral infarct size in cerebral ischemia/reperfusion mouse models[J]. Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi,2016,32(3):294-298.
[6]	Zeng S,Zhou X,Ge L, et al. Monocyte subsets and monocyte-platelet aggregates in patients with unstable angina[J]. Journal of thrombosis and thrombolysis, 2014, 38 (4): 439-446.
[7]	Zhou X,Liu XL,Ji WJ, et al. The Kinetics of circulating monocyte subsets and monocyte-platelet aggregates in the acute phase of ST-Elevation myocardial infarction: associations with 2-Year cardiovascular events[J]. Medicine, 2016, 95 (18): e3466-e3475.
[8]	Zhou X,Zhang L,Ji W J, et al. Variation in dietary salt intake induces coordinated dynamics of monocyte subsets and monocyte-platelet aggregates in humans: implications in end organ inflammation[J]. PLoS One,2013,8(4):e60332-e60344.
[9]	Barbosa R M,Speretta G F,Dias D P, et al. Increased expression of macrophage migration inhibitory factor in the nucleus of the solitary tract attenuates renovascular hypertension in rats[J]. Am J Hypertens,2017, 30 (4): 435-443.
[10]	DuBrock H M,Rodriguez-Lopez J M,LeVarge B L, et al. Macrophage migration inhibitory factor as a novel biomarker of portopulmonary hypertension[J]. Pulm Circ,2016,6(4):498-507.
[11]	Harwani S C,Ratcliff J,Sutterwala F S, et al. Nicotine mediates CD161a+renal macrophage infiltration and premature hypertension in the spontaneously hypertensive rat[J]. Circ Res,2016,119(10):1101-1115.
[12]	Meng X M,Tang P M,Li J, et al. Macrophage phenotype in kidney injury and repair[J]. Kidney Dis (Basel),2015,1(2):138-146.
[13]	Bruchfeld A,Wendt M, Miller E J. Macrophage migration inhibitory factor in clinical kidney disease[J]. Front Immunol,2016,7(1): 1-7.
[14]	Tokuda K, Kai H, Kuwahara F, et al. Pressure-independent effects of angiotensin II on hypertensive myocardial fibrosis[J]. Hypertension, 2004, 43 (2): 499-503.
[15]	Ogata T, Miyauchi T, Sakai S, et al. Myocardial fibrosis and diastolic dysfunction in deoxycorticosterone acetate-salt hypertensive rats is ameliorated by the peroxisome proliferator-activated receptor-alpha activator fenofibrate, partly by suppressing inflammatory responses associated with the nuclear factor-kappa-B pathway[J]. J Am Coll Cardiol, 2004, 43 (8): 1481-1488.
[16]	Damilano F, Franco I, Perrino C, et al. Distinct effects of leukocyte and cardiac phosphoinositide 3-kinase gamma activity in pressure overload-induced cardiac failure[J]. Circulation, 2011, 123 (4): 391-399.
[17]	Huang X R, Chung A C, Yang F, et al. Smad3 mediates cardiac inflammation and fibrosis in angiotensin II-induced hypertensive cardiac remodeling[J]. Hypertension, 2010, 55 (5): 1165-1171.
[18]	Machnik A,Neuhofer W,Jantsch J, et al. Macrophages regulate salt-dependent volume and blood pressure by a vascular endothelial growth factor-C-dependent buffering mechanism[J]. Nat Med,2009,15(5):545-552.
[19]	Titze J, Machnik A. Sodium sensing in the interstitium and relationship to hypertension[J]. Curr Opin Nephrol Hypertens,2010,19(4):385-392.
[20]	Machnik A,Dahlmann A,Kopp C, et al. Mononuclear phagocyte system depletion blocks interstitial tonicity-responsive enhancer binding protein/vascular endothelial growth factor C expression and induces salt-sensitive hypertension in rats[J].Hypertension,2010,55(3):755-761.
[21]	Gao F,Han ZQ,Zhou X, et al. High salt intake accelerated cardiac remodeling in spontaneously hypertensive rats: time window of left ventricular functional transition and its relation to salt-loading doses[J]. Clin Exp Hypertens,2011,33(7):492-499.
[22]	Yang G H,Zhou X,Ji WJ, et al. Overexpression of VEGF-C attenuates chronic high salt intake-induced left ventricular maladaptive remodeling in spontaneously hypertensive rats[J]. Am J Physiol Heart Circ Physiol,2014,306(4):H598-609.
[23]	Hogan B M, Black B L. Developmental biology: diversity in the lymphatic vasculature[J]. Nature,2015,522(7554):37-38.
[24]	Kim K W, Song J H. Emerging roles of lymphatic vasculature in immunity[J]. Immune Netw,2017,17(1):68-76.
[25]	Cui Y. Impact of lymphatic vessels on the heart[J]. Thorac Cardiovasc Surg,2010,58(1):1-7.
[26]	Sun Q N,Wang Y F, Guo Z K. Reconstitution of myocardial lymphatic vessels after acute infarction of rat heart[J]. Lymphology,2012,45(2):80-86.
[27]	Rockson S G. Lymphatics and the heart: the importance of visceral lymphatic function in health and disease[J]. Lymphat Res Biol,2007,5(1):1-2.
[28]	Li X,Shimada T,Zhang Y, et al. Ultrastructure changes of cardiac lymphatics during cardiac fibrosis in hypertensive rats[J]. Anat Rec (Hoboken),2009,292(10):1612-1618.
[29]	Cursiefen C,Chen L,Borges L P, et al. VEGF-A stimulates lymphangiogenesis and hemangiogenesis in inflammatory neovascularization via macrophage recruitment[J]. J Clin Invest,2004,113(7):1040-1050.
[30]	Kerjaschki D,Regele H M,Moosberger I, et al. Lymphatic neoangiogenesis in human kidney transplants is associated with immunologically active lymphocytic infiltrates[J]. J Am Soc Nephrol,2004,15(3):603-612.
[31]	Schoppmann S F,Horvat R, Birner P. Lymphatic vessels and lymphangiogenesis in female cancer: mechanisms, clinical impact and possible implications for anti-lymphangiogenic therapies (Review)[J]. Oncol Rep,2002,9(3):455-460.
[32]	Maruyama K,Ii M,Cursiefen C, et al. Inflammation-induced lymphangiogenesis in the cornea arises from CD11b-positive macrophages[J]. J Clin Invest,2005,115(9):2363-2372.
[33]	Kerjaschki D. The crucial role of macrophages in lymphangiogenesis[J]. J Clin Invest,2005,115(9):2316-2319.