Folkestad T, Brurberg K G, Nordhuus K M, et al. Acute kidney injury in burn patients admitted to the intensive care unit: a systematic review and meta-analysis[J]. Crit Care, 2020, 24(1): 2.
[2]
Ysng G, Tan L, Yao H, et al. Long-Term Effects of severe burns on the kidneys: research advances and potential therapeutic approaches[J]. J Inflamm Res, 2023, 16(1):1905-1921.
[3]
Wainstein M, Macdonald S, Fryer D, et al. Use of an extended KDIGO definition to diagnose acute kidney injury in patients with COVID-19: A multinational study using the ISARIC-WHO clinical characterisation protocol[J]. PLoS Med, 2022, 19(4): e1003969.
[4]
Legrand M, Kellum J A. Serum creatinine in the critically Ill patient with sepsis[J]. JAMA, 2018, 320(22): 2369-2370.
[5]
Ostermann M, Zarbock A, Goldstein S, et al. Recommendations on acute kidney injury biomarkers from the acute disease quality initiative consensus conference: a consensus statement[J]. JAMA Netw Open, 2020, 3(10): e2019209.
[6]
Legrand M, Clark A T, Neyra J A, et al. Acute kidney injury in patients with burns[J]. Nat Rev Nephrol, 2024, 20(3): 188-200.
[7]
Soussi S, Dépret F, Benyamina M, et al. Early hemodynamic management of critically ill burn patients[J]. anesthesiology, 2018, 129(3): 583-589.
[8]
Tran N K, Sen S, Palmieri T L, et al. Artificial intelligence and machine learning for predicting acute kidney injury in severely burned patients: a proof of concept[J]. Burns, 2019, 45(6): 1350-1358.
[9]
Ren H, Zhou X, Dai D, et al. Assessment of urinary kidney injury molecule-1 and interleukin-18 in the early post-burn period to predict acute kidney injury for various degrees of burn injury[J]. BMC Nephrol, 2015, 16(18): 142.
[10]
Khorashadi M, Beunders R, Pickkers P, et al. Proenkephalin: a new biomarker for glomerular filtration rate and acute kidney injury[J]. Nephron, 2020, 144(12): 655-661.
[11]
Dépret F, Polina A, Amzallag J, et al. PenKid measurement at admission is associated with outcome in severely ill burn patients[J]. Burns, 2020, 46(6): 1302-1309.
[12]
Chi Y, Liu X, Chai J. A narrative review of changes in microvascular permeability after burn[J]. Ann Transl Med, 2021, 9(8): 719.
Wang M, Scott S R, Koniaris L G, et al. Pathological responses of cardiac mitochondria to burn trauma[J]. Int J Mol Sci, 2020, 21(18): 6655.
[15]
Dudoignon E, Dépret F, Legrand M. Is the Renin-angiotensin-aldosterone system good for the kidney in acute settings?[J]. Nephron, 2019, 143(3): 179-183.
[16]
Ko A, Song J, Golovko G, et al. Higher risk of acute kidney injury and death with rhabdomyolysis in severely burned patients[J]. Surgery, 2022, 171(5): 1412-1416.
[17]
Chen B, Zhao J, Zhang Z, et al. Clinical characteristics and risk factors for severe burns complicated by early acute kidney injury[J]. Burns, 2020, 46(5): 1100-1106.
[18]
Khorashadi M, Bokoch M P, Legrand M. Is nitric oxide the forgotten nephroprotective treatment during cardiac surgery?[J]. Ann Intensive Care, 2020, 10(1): 22.
[19]
Jeschke M G, Van Baar M E, Choudhry M A, et al. Burn injury[J]. Nat Rev Dis Primers, 2020, 6(1): 11.
[20]
Comish P B, Carlson D, Kang R, et al. Damage-associated molecular patterns and the systemic immune consequences of severe thermal injury[J]. J Immunol, 2020, 205(5): 1189-1197.
[21]
Mulder P P G, Vlig M, Boekema B, et al. Persistent systemic inflammation in patients with severe burn injury is accompanied by influx of immature neutrophils and shifts in t cell subsets and cytokine profiles[J]. Front Immunol, 2020, 11(29): 621222.
[22]
Guo S, Yu M, Fang Q, et al. Heme oxygenase-1 induction mitigates burn-associated early acute kidney injury via the TLR4 signaling pathway[J]. Burns, 2022, 48(1): 156-167.
[23]
Guo S, Guo L, Fang Q, et al. Astaxanthin protects against early acute kidney injury in severely burned rats by inactivating the TLR4/MyD88/NF-κB axis and upregulating heme oxygenase-1[J]. Sci Rep, 2021, 11(1): 6679.
[24]
Liu X, Liu Z, Li D, et al. Mitochondria play a key role in oxidative stress-induced pancreatic islet dysfunction after severe burns[J]. J Trauma Acute Care Surg, 2022, 92(6): 1012-1019.
Herrler T, Tischer A, Meyer A, et al. The intrinsic renal compartment syndrome: new perspectives in kidney transplantation[J]. Transplantation, 2010, 89(1): 40-46.
[27]
Calfee C S, Delucchi K L, Sinha P, et al. Acute respiratory distress syndrome subphenotypes and differential response to simvastatin: secondary analysis of a randomised controlled trial[J]. Lancet Respir Med, 2018, 6(9): 691-698.
[28]
Dépret F, Hoffmann C, Daoud L, et al. Association between hydroxocobalamin administration and acute kidney injury after smoke inhalation: a multicenter retrospective study[J]. Crit Care, 2019, 23(1): 421.
[29]
Evans J, Pandya A, Ding Y, et al. Hydroxocobalamin-induced oxalate nephropathy in a patient with smoke inhalation[J]. Kidney Int Rep, 2021, 6(8): 2228-2231.
[30]
Kıroglu O E, Özü Ö Y, Emre M, et al. Residual NO modulates contractile responses and membrane potential in isolated rat mesenteric arteries[J]. Nitric Oxide, 2017, 71(1): 21-26.
[31]
You B, Yang Z, Zhang Y, et al. Late-onset acute kidney injury is a poor prognostic sign for severe burn patients [J]. Front Surg, 2022, 9(2): 842999.
[32]
Schult L, Halbgebauer R, Karasu E, et al. Glomerular injury after trauma, burn, and sepsis[J]. J Nephrol, 2023, 36(9): 2417-2429.
Toporek A H, Semler M W, Self W H, et al. Balanced crystalloids versus saline in critically ill adults with hyperkalemia or acute kidney injury: secondary analysis of a clinical trial[J]. Am J Respir Crit Care Med, 2021, 203(10): 1322-1325.
[35]
Hammond N E, Zampieri F G, Di Tanna G L, et al. Balanced crystalloids versus saline in critically ill adults-a systematic review with meta-analysis[J]. NEJM Evid, 2022, 1(2): EVIDoa2100010.
[36]
Ostermann M, Bagshaw S M, Lumlertgul N, et al. Indications for and Timing of Initiation of KRT[J]. Clin J Am Soc Nephrol, 2023, 18(1): 113-120.
[37]
Lavrentieva A, Depetris N, Moiemen N, et al. Renal replacement therapy for acute kidney injury in burn patients, an international survey and a qualitative review of current controversies[J]. Burns, 2022, 48(5): 1079-1091.
[38]
Digvijay K, Neri M, Fan W, et al. International survey on the management of acute kidney injury and continuous renal replacement therapies: year 2018[J]. Blood Purif, 2019, 47(1-3): 113-119.
[39]
Liang K V, Sileanu F E, Clermont G, et al. Modality of rrt and recovery of kidney function after aki in patients surviving to hospital discharge[J]. Clin J Am Soc Nephrol, 2016, 11(1): 30-38.
[40]
Tan B K, Liew Z H, Kaushik M, et al. Early Initiation of renal replacement therapy among burned patients with acute kidney injury[J]. Ann Plast Surg, 2020, 84(4): 375-378.
[41]
Wald R, Beaubien-Souligny W, Chanchlani R, et al. Delivering optimal renal replacement therapy to critically ill patients with acute kidney injury[J]. Intensive Care Med, 2022, 48(10): 1368-1381.
[42]
Ostermann M, Bellomo R, Burdmann E A, et al. Controversies in acute kidney injury: conclusions from a kidney disease: improving global outcomes (KDIGO) conference[J]. Kidney Int, 2020, 98(2): 294-309.
[43]
Fahmy S R, Soliman A M, El Ansary M, et al. Therapeutic efficacy of human umbilical cord mesenchymal stem cells transplantation against renal ischemia/reperfusion injury in rats[J]. Tissue Cell, 2017, 49(3): 369-375.
[44]
Fazekas B, Alagesan S, Watson L, et al. comparison of single and repeated dosing of anti-inflammatory human umbilical cord mesenchymal stromal cells in a mouse model of polymicrobial sepsis[J]. Stem Cell Rev Rep, 2022, 18(4): 1444-1460.
[45]
Hu Q, Lyon C J, Fletcher J K, et al. Extracellular vesicle activities regulating macrophage- and tissue-mediated injury and repair responses[J]. Acta Pharm Sin B, 2021, 11(6): 1493-1512.
[46]
Wan F, Yang R C, Tang Y W, et al. BMSC-derived exosomes protect against kidney injury through regulating klotho in 5/6 nephrectomy rats[J]. Eur J Med Res, 2022, 27(1): 118.
[47]
Huang J, Cao H, Cui B, et al. Mesenchymal stem cells-derived exosomes ameliorate ischemia/reperfusion induced acute kidney injury in a porcine model[J]. Front Cell Dev Biol, 2022, 10(24): 899869.
[48]
Cao J Y, Wang B, Tang T T, et al. Exosomal miR-125b-5p deriving from mesenchymal stem cells promotes tubular repair by suppression of p53 in ischemic acute kidney injury [J]. Theranostics, 2021, 11(11): 5248-5266.
2025, Vol. 36 
